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Collins’ group theorizes that if this effect could be amplified, or if the cell’s genetic defense against it can be weakened, no bacterias could withstand its impact and the emergence of antibiotic-resistant bacteria could be limited.’ But, there’s actually nothing incorrect. Previous function by Kohanski and co-lead author Dan Dwyer, a postdoctoral researcher in Collins’ lab, revealed the initial hints that underlying pathway exists. In studying bacterial response to a quinolone, an antibiotic that inhibits DNA replication, they noted a surprising change in genes in charge of energy iron and production uptake.